Search results for "Toll-Like Receptor 5"

showing 4 items of 4 documents

Successive Losses of Central Immune Genes Characterize the Gadiformes' Alternate Immunity.

2016

Great genetic variability among teleost immunomes, with gene losses and expansions of central adaptive and innate components, has been discovered through genome sequencing over the last few years. Here, we demonstrate that the innate Myxovirus resistance gene (Mx) is lost from the ancestor of Gadiformes and the closely related Stylephorus chordatus, thus predating the loss of Major Histocompatibility Complex class II (MHCII) in Gadiformes. Although the functional implication of Mx loss is still unknown, we demonstrate that this loss is one of several ancient events appearing in successive order throughout the evolution of teleost immunity. In particular, we find that the loss of Toll-like r…

0106 biological sciences0301 basic medicineFish ProteinsLineage (genetic)LetterGenes MHC Class IIZoologyParacanthopterygiiadaptationteleosts010603 evolutionary biology01 natural sciencesEvolution Molecular03 medical and health sciencesOrthomyxoviridae InfectionsPhylogeneticsGeneticsAnimalsGenetic variabilityGeneinnate immunityEcology Evolution Behavior and SystematicsInnate immune systemPolymorphism GeneticbiologyGadiformesadaptive immunitygene lossAcquired immune systembiology.organism_classificationGadiformesToll-Like Receptor 5030104 developmental biologyEvolutionary biologyMyxovirus resistance (Mx)Gene DeletionGenome biology and evolution
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Toll-like receptor 5 deficiency exacerbates cardiac injury and inflammation induced by myocardial ischaemia-reperfusion in the mouse

2015

Myocardial ischaemia-reperfusion (MIR) triggers a sterile inflammatory response important for myocardial healing, but which may also contribute to adverse ventricular remodelling. Such inflammation is initiated by molecular danger signals released by damaged myocardium, which induce innate immune responses by activating toll-like receptors (TLRs). Detrimental roles have been recently reported for TLR2, TLR3 and TLR4. The role of other TLRs is unknown. We therefore evaluated the role of TLR5, expressed at high level in the heart, in the development of myocardial damage and inflammation acutely triggered by MIR. TLR5−/− and wild-type (WT) mice were exposed to MIR (30 min ischaemia, 2 h reperf…

MaleChemokinemedicine.medical_specialtyGenotypep38 mitogen-activated protein kinasesMyocardial InfarctionMyocardial Reperfusion InjuryInflammation030204 cardiovascular system & hematologyBiologyp38 Mitogen-Activated Protein KinasesVentricular Function LeftProinflammatory cytokineVentricular Dysfunction Left03 medical and health sciences0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicinemedicineAnimalsPhosphorylationProtein kinase B030304 developmental biologyInflammationMice Knockout0303 health sciencesToll-like receptorMyocardiumGeneral MedicineImmunity Innate3. Good healthMice Inbred C57BLDisease Models AnimalOxidative StressToll-Like Receptor 5CXCL2PhenotypeEndocrinologybiology.proteinTLR4Inflammation Mediatorsmedicine.symptomProto-Oncogene Proteins c-aktClinical Science
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Predictive value of p53, Ki67 and TLR5 in neoplastic progression of Barrett’s esophagus: a matched case–control study

2022

Abstract Barrett’s esophagus progresses to high-grade dysplasia or cancer along the well-established metaplasia-dysplasia-adenocarcinoma sequence. The aim of this study was to evaluate the value of p53, Ki67, and toll-like receptor 5 (TLR5) in prediction of malignant progression of Barrett’s metaplasia and low-grade dysplasia. This was a retrospective matched case–control study based on Northern and Central Finland population. Patients diagnosed with esophageal high-grade dysplasia or adenocarcinoma were included. From these patients, all previous endoscopy samples were obtained along with original diagnostic HE-slides and clinical data. Age- and sex-matched patients with non-progressing Ba…

p53Esophageal NeoplasmsbiomarkkeritAdenocarcinomaPathology and Forensic MedicineimmunohistokemiaBarrett EsophagusHumansBarrett’s esophagusTLR5Molecular BiologyRetrospective StudiesMetaplasiaSurveillanceCell BiologyGeneral MedicineImmunohistochemistrydigestive system diseasesToll-Like Receptor 5antigeenitKi-67 AntigenDysplasia markerCase-Control StudiesproteiinitEsophageal adenocarcinomatarkkailuTumor Suppressor Protein p53ruokatorvisyöpäKi67Virchows Archiv
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Enterobacter cloacae administration induces hepatic damage and subcutaneous fat accumulation in high-fat diet fed mice.

2018

Accumulating evidence indicates that gut microbiota plays a significant role in obesity, insulin resistance and associated liver disorders. Family Enterobacteriaceae and especially Enterobacter cloacae strain B29 have been previously linked to obesity and hepatic damage. The underlying mechanisms, however, remain unclear. Therefore, we comprehensively examined the effects of E. cloacae subsp. cloacae (ATCC® 13047™) administration on host metabolism of mice fed with high-fat diet (HFD). C57BL/6N mice were randomly divided into HFD control, chow control, and E. cloacae treatment groups. The E. cloacae treatment group received live bacterial cells in PBS intragastrically twice a week, every ot…

rasvahapotPathology and Laboratory Medicinerasvat (orgaaniset yhdisteet)ruokavaliotBiochemistryMiceAnimal CellsFibrosislcsh:ScienceImmune ResponseConnective Tissue CellsChemical Reactionsta3141ta3142Lipids3. Good healthPhysical sciencesAdiponectinCellular Typesmedicine.medical_specialtyfatsLipolysisImmunologySubcutaneous FatrasvakudoksetMonomers (Chemistry)glycerolDiet High-Fatta311103 medical and health sciencesSigns and SymptomsEnterobacter cloacaeLipolysisPolymer chemistrylcsh:RBiology and Life SciencesHypertrophymedicine.diseaseReceptor InsulinMice Inbred C57BLBiological Tissue030104 developmental biologyEndocrinologylihavuuslcsh:QGlycerol0301 basic medicinePhysiologyLiver cytologysuolistomikrobistolcsh:MedicineAdipose tissueGut floraMedicine and Health SciencesAdipocytesenterobakteerit2. Zero hungerrasvatMultidisciplinarygastrointestinal microbiotatulehdusbiologyHydrolysisadipose tissueChemistryPhysiological ParametersLiverConnective Tissueembryonic structuresFemaleAnatomymedicine.symptomResearch Articleanimal structuresadipocytesInflammationInsulin resistanceEnterobacteriaceaeDiagnostic MedicineInternal medicinemedicineAnimalsObesityTriglyceridesNutritionurogenital systembusiness.industryBody WeightCell Biologybiology.organism_classificationDietToll-Like Receptor 5Gene Expression RegulationinflammationlipolysisdietbusinessEnterobacter cloacaePLoS ONE
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